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Description
Childhood ALL is often polyclonal at diagnosis and under the selective influence of therapy, some clones may be extinguished and new clones with distinctive genomic profiles may arise.[13] However, molecular subtypedefining lesions such as translocations and aneuploidy are almost always retained at relapse.[1,13] Of particular importance are new variants that arise at relapse that may be selected by specific components of therapy
Gontero, W
PMID: 25746857 Cervellati C, Bergamini CM
Attanasio, A
Nutrients, 8(12), 803
