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PD patient-derived S triplication neurons have abnormal pSer129 S and UPR levels and decreased S T:M phenotypes, all of which are reversed by LIPE inhibition E46K S or wt S overexpression models are premised on the mutated or excess S interacting more or differently with vesicular phospholipid membranes
They are also relatively inexpensive and easy to install
doi: 10.1111/cei.13266
The genetic inactivation of inducible nitric oxide synthase (iNOS) intensifies fibrosis and oxidative stress in the penile corpora cavernosa in type 1 diabetes
CXCL12/CXCR4 signalling activates MMP-9 expression in prostate cancer cells [43]
