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Finally, we outline the implications of adopting semaglutide as a disease-modifying agent in MASH with F2-F3 fibrosis, note the study limitations and remaining questions, and argue that combinatorial approaches targeting complementary metabolic and hepatocentric pathways may define the next phase of MASLD management

effects on liver steatosis, hepatocyte ballooning, inflammation, fibrosis (F1-F3), cirrhosis (F4), treatment duration, and safety (treatment-emergent adverse events)

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