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Researchers are still learning about their other potential uses
[16] These medications work through multiple complementary mechanisms: they enhance glucose-dependent insulin secretion from pancreatic beta cells, suppress inappropriate glucagon release from alpha cells, slow gastric emptying to reduce postprandial glucose excursions, and act on central nervous system pathways to promote satiety and reduce appetite
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Abstract 54-OR
Systolic blood pressure (SBP) was significantly of lowest measures in the control group versus all GLP1-RA groups (p = 0.027)
