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The insulin sensitization and hepatic glucose output reduction from berberine are independent of GLP-1 receptor agonism, which means they persist after tirzepatide is removed

to map clinical heterogeneity in T2D to individual etiologic processes using soft clustering of 32 anthropometric, clinical, and biochemical phenotypes in early T2D identified 4 archetypes based on obesity, insulin resistance, dyslipidemia, and -cell glucose sensitivity [26]