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DHEAS was protective against the neurotoxin MPP+ (which inhibits catecholamine synthesis and triggers cell death) in rat cerebellar granule cell cultures [138]
It is a metabolic intervention
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This feedback loop exacerbates the loss of GRAIL and amplifies mTOR activation, thereby impairing Treg function and promoting immune dysregulation
Systolic BP remained stable during the maintenance phase for those assigned semaglutide and increased for those assigned placebo (difference, 3.9 mm Hg
