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GIP is best known for its ability to act on islets, where it regulates blood glucose levels by stimulating the secretion of insulin or glucagon (Miyawaki et al., 2002)

Produces b-ions and y-ions Provides amino acid sequence information Enables localization of modifications Key Insight: Optimized fragmentation conditions are critical for complete sequence coverage

The primary endpoint will be HbA1c change

Both GIP (186) and GLP-1 (187) enhance adiponectin expression and secretion in murine models

While IGF-1 has insulin-agonistic actions and has been used for treatment of severe insulin resistance, IGF-1 reduction with the GH antagonist pegvisomant improves glycemic control in acromegalic patients with diabetes or impaired glucose tolerance despite elevated GH (35, 36)