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Tanelian and Monroe (37) studied a rabbit model in which they produced corneal nerve fiber injury by a small punch biopsy and subsequently used electrophysiological methods to directly determine the behavior of regenerating small nerve fibers to cold stimuli

In the context of renal injury, GM-CSF, LPS, IL-1, TNF-, FN-, and various endogenous damage-associated molecular patterns (DAMPs) released by the damaged renal cells promote the polarization of macrophages to the M1 phenotype (6567)

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