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Consistent with a disabled lipid peroxidation defense, we observed that after one hour of GPX4 inhibition and prior to any cell death, there is a drastic elevation in 2,7-dichlorofluorescein (DCF) staining of ROS which indicates lipid peroxidation in persister cells but not parental cells (Fig

found in their study that in clear cell renal cell carcinoma (ccRCC) tumor cells, endogenous Gln metabolism might reduce the anti-tumor effectiveness of T cells by promoting the Treg cell response ( 2.1.4 Regulation of tumor cell redox homeostasis Gln regulates the redox status of cancer cells by influencing the production of mitochondrial ROS

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