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Taken together, these studies underscore the therapeutic potential of iPSC-derived neural progenitors in PD models, yet the variability in reprogramming methods reported in the aforementioned studies, limited scalability, and unresolved concerns around long-term safety and functional integration highlight the need for more standardized and clinically validated approaches

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Downregulated CD36 and oxLDL uptake and stimulated ABCA1/G1 and cholesterol efflux as anti-atherosclerotic mechanisms of interleukin-10

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