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This intrinsic anti-ferroptosis mechanism allows M1 macrophages to survive in the pro-inflammatory environment and continuously perform immune surveillance functions, constituting their unique cellular identity (148, 149)

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[DOI] [PMC free article] [PubMed] [Google Scholar] 381.Palmisano B, Labella R, Donsante S, Remoli C, Spica E, Coletta I, Farinacci G, Dello Spedale VM, Saggio I, Serafini M, Robey PG, Corsi A, Riminucci M

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