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Synthetic approaches to protein phosphorylation

In postmortem studies of COVID-19, significantly higher levels of senescent cells have been observed in multiple tissues, including the brain

A recent study by Wei, et al

It is postulated that somatic loss of the remaining wild-type allele in the tumor cells would lead to a homologous repair deficient (HRD) phenotype, rendering such tumors exquisitely sensitive to agents that induce double strand DNA breaks (e.g., platinum agents like cisplatin and oxaliplatin), or to inhibitors of the DNA repair enzyme PARP, which would result in synthetic lethality given the pre-existing compromise in DNA repair function 86

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