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Pathophysiological Mechanisms Underlying IUA Central Pathological Feature: Tissue Fibrosis Activation of Fibroblasts and Myofibroblast Transdifferentiation The hallmark pathological feature of intrauterine adhesion (IUA) is endometrial fibrosis, primarily characterized by the aberrant deposition of extracellular matrix (ECM) components such as collagen

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ALCAR administration induced mitochondrial biogenesis in hypoxic rats [15], and increased mitochondrial mass after spinal cord injury [13]

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