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Phase 2 subclassifies patients into notional mechanistic subtypes (neuroinflammation-dominant, excitotoxicity-dominant, oxidative-stress-dominant, or neurotrophic-epigenetic-dominant) based on the predominant biomarker signatures, a concept directly derived from the stratification logic described in this section

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Ferroptosis signaling promotes functional recovery after sciatic nerve injury in mice Although axonal fusion after injury has not been directly demonstrated in vertebrates to date, there is extensive evidence showing that axonal fusion-like mechanisms can be induced to promote nerve repair in mammals, including humans 1

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