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As a summary, our model suggests acetylation of SnCE1 at Lys231 is catalysed by irreversible autoacetylation and a mechanism to increase SnCE1 deSUMOylase activity by shifting the tetramer-monomer equilibrium towards the SnCE1 monomeric form

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Therefore, the ideal treatment combination needs to be identified for each CRC patient

Dose-dependent accumulation of ET in the eye could suggest an important role in providing protection, since oxidative stress in the eye is common in ocular diseases 50,51

The unique ability of the BBB to sequester GSH from the blood circulation through its Na + -dependent GSH transporter provides a solid foundation for using the transporter as a target to develop brain-targeting drug delivery systems