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Description
Gram-negative bacterial cell wall lipopolysaccharide (LPS) activates Toll-like receptors 4 (TLR4) on the surface of intestinal epithelial cells and mediates enhanced intestinal permeability, the activation of TLR in plasma and brain cells by LPS can induce the release of pro-inflammatory cytokines and lead to memory deficits (Yang et al., 2020b)
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Moreover, current therapies can cause severe long-term side effects and new therapies are urgently needed
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