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This treatment decreased iNOS, oxidative stress, and neuroinflammation without activating CB1/CB2 receptors, highlighting CBDs antioxidant and anti-inflammatory properties in HD progression (Sagredo et al

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PMID: Summary Keywords drug-induced liver injury, StevensJohnson syndrome, toxic epidermal necrolysis, immune checkpoint inhibitors, moxifloxacin Citation Li Q, Wang L, Xu H, Huang T, Hong H, Ji X and Liu J (2026) Concurrent moxifloxacin-induced liver injury and toxic epidermal necrolysis after immune checkpoint inhibition: a case report and literature review

Ly6Chi Monocytes and Their Macrophage Descendants Regulate Neutrophil Function and Clearance in Acetaminophen-Induced Liver Injury