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Our immunologic findings support the hypothesis that persistent viral antigens or tissue damage may promote lingering innate immune responses and systemic low-grade inflammation following SARS-CoV-2 infection, although it remains unclear to what extent this is mechanistically tied to LC symptoms

Mitochondria-dependent apoptosis was involved in the alleviation of Jujuboside A on diabetic kidney disease-associated renal tubular injury via YY1/PGC-1 signaling

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