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The lack of MRS dACC changes in the Recent onset group in this or other studies does not amount to a decisive refutation of the glutamate neurotoxicity hypothesis because the relationship of static MRS glutamate to synaptic status is too uncertain and the time of onset of the putative glutamate excess relative to psychosis onset is unknown

Clinical Pharmacology & Therapeutics

Necrosis has an important association with the pathological mechanism of OA, but further clinical studies are needed to confirm the causal relationship between them

59 Scientific evidence indicates that not necessarily the deficiency but the low status of the B 12 biomarker has been associated with increased total Hcy level, which is involved in increased generation of reactive oxygen species in lipid peroxidation and tissue damage to the endothelium vascular and thromboembolism

doi:10.1002/jat.736