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funded the experimental work
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The ECAR data mirrored these findings as further confirmation of reduced oxidative phosphorylation activity (Fig
A significant decrease in adenosine triphosphate (ATP) levels as a result of age and elevated IOP in the optic nerve, as well as a correlation between the rate of axonal dysfunction and the depletion of energy substrates as a result of oxygenglucose deprivation, further implicate metabolism in glaucoma pathogenesis (Baltan et al., 2010)
