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However, our multi-platform approach provided a more comprehensive metabolomic profile, revealing additional potential biomarkers and metabolic pathways involved in GDM pathogenesis

When TSC1/TSC2 is phosphorylated by AKT1 it is less effective at stimulating the GTPase activity of Rheb and therefore Rheb activation of mTOR will remain high as is the case in response to insulin action

This suggests that additional dietary modifications, such as methionine restriction, may be needed to modulate neuroinflammation more effectively

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[DOI] [PubMed] [Google Scholar] Littleton J (1998) Neurochemical mechanisms underlying alcohol withdrawal