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When expressing URA3 heterozygously at the GSH1 locus, a loss of heterozygosity became evident over time under 5-FOA selection, likely attributable to mitotic crossovers

coli measured in media supplemented with 1 g/L glucose (EGlc) at 0 and 24 h, respectively

Furthermore, the density of ANFs passing through the habenula perforata in the apical, middle, and basal turns of the cochlea was more severely reduced in SIRT1 CKO + noise mice than in SIRT1 flox/flox + noise mice following noise exposure (Fig

It suggested that NF-B p65 may be a potential therapeutic target for UC
