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Key actions include: Activation of the folate-polyamine-AICAR pathway, triggering AMPK signaling independently of cellular energy depletion Enhanced glucose uptake in skeletal muscle via GLUT4 translocation, resembling insulin-responsive metabolic pathways Interaction with nuclear transcription factors to support antioxidant defense and control of inflammatory pathways Through these mechanisms, MOTS-c functions as a mitochondria-to-nucleus messenger, facilitating cellular adaptation to energy demands and oxidative stress

Additionally, we systematically analyzed the natural bioactive compounds demonstrating neuroprotective efficacy in preclinical stroke models

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10.1016/j.theriogenology.2010.04.023 40 de SouzaA

95% CI -30.53 to 7.53) (p=0.04) when compared to placebo