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academic.oup.com, doi:10.3945/ajcn.110.003483

Researchers frequently examine its interaction with growth factor signaling and cellular repair mechanisms

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This suggestion was further insisted as a potential mechanism of TB-500 by the authors of the research study, who also commented that "transfection of anti-miR-146a nucleotides reversed the inhibitory effect of T4 on IRAK1 and TRAF6." Thus, TB-500 also appears to potentially work for healing by exerting anti-inflammatory action.(6)

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