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Gram-negative bacterial cell wall lipopolysaccharide (LPS) activates Toll-like receptors 4 (TLR4) on the surface of intestinal epithelial cells and mediates enhanced intestinal permeability, the activation of TLR in plasma and brain cells by LPS can induce the release of pro-inflammatory cytokines and lead to memory deficits (Yang et al., 2020b)

Numerous other studies have also chosen this mouse model as one of the best characterized and most informative mouse models for TDP-43 pathology 39,40,41,42,43

Yu et al., 2021) and Wang et al

Sample values are clearly marked as demo records

7a, b), suggesting that RBM15 SPOC might be a more promiscuous phosphoserine binding domain