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doi: 10.1001/jama.1960.03030100059013 242 SullivanMPBeattyECHymanCBMurphyMLPierceMISeveroNC
Nonetheless, targeting this population is anticipated to provide new treatment opportunities
Wu C, Duan F, Yang R et al (2023) 15, 16-Dihydrotanshinone I protects against ischemic stroke by inhibiting ferroptosis via the activation of nuclear factor erythroid 2-related factor 2
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Metabolic reprogramming, including enhanced polyunsaturated fatty acid (PUFA) synthesis and glycolytic flux, provides the essential substrates and energy to sustain this vicious cycle
