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Description
It helps transport fatty acids to the muscles mitochondria so they can be used as energy
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when cells are exposed to this stimulus, the NRF2Keap1 complex is disrupted, allowing NRF2 to escape and resulting in Keap1-mediated degradation
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The study result indicated its most likely because of new protein synthesis and the activation of the mTOR pathway (a driver for increased muscle mass)
How It Fits the SHARP Protocol
