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For example, ADCC mediated by daratumumab against primary MM cells was 14.2% in cells within the lowest tertile of CD38 expression vs

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The VEGFR2-Akt-eNOS signaling pathway was directly activated (i.e., without the need for other known ligands or shear stress), controlling vasomotor tone (both smooth muscle and endothelium), and the Src-Caveolin-1-eNOS pathway was activated [103,104]