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Description
Final concentration 5 mg/ml
The dysregulation of miR-103 may be a contributing factor for abnormal fatty acid metabolism in ASD, and miR-146a defects may lead to impaired phagocytosis in immune responses
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AO can interact with metabolic Glu receptor 5 to promote long-term depression and inhibit long-term potentiation in the hippocampus, leading to downstream responses and kinases activation
Breitholtz-Emanuelsson A
