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To test the effect of sustained repression of lipogenic programs and a concurrent sustained induction of NRF2 activity, we monitored mice for ten weeks following the induction of Gclc deletion alone (Gclc L-KO) or in combination with Nrf2 deletion (L-DKO) in the liver (Fig

Anabolic hormonal signaling, including testosterone, growth hormone, and IGF-1

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L4-L5 herniations The L4-L5 junction is one of the most mobile segments of the lumbar spine, making it particularly vulnerable to disc herniation

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