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Similarly, Th2 cells, which are responsible for promoting humoral immunity and antibody production, are disrupted in ME/CFS [55]

Since dysregulation within these pathways is frequently associated with organ-specific damage in experimental settings, the data suggest that BPC-157 may support signaling balance by attenuating excessive activation or mitigation within these interconnected systems

Furthermore, progressive loss of NRF2 activity was seen with aging, which may be explained by studies showing that the expression of NRF2 positive regulators P62 and BRCA1 decreased with age, while NRF2 negative regulators, KEAP1 and BACH1, increased with age [113]