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Description
Identification of a dominant epitope of glutamic acid decarboxylase (GAD-65) recognized by autoantibodies in stiff-man syndrome

In essence, MG significantly influences atherosclerosis onset through both enhanced levels and compromised GLO I detoxification, positioning it as a potential therapeutic target for comprehensive intervention [79]

These studies were generally normal
The evidence for clinically meaningful superiority over regular glutathione is not yet established [1]

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