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CGA can attenuate alcohol-induced pathologies such as steatosis, apoptosis, and fibrosis by regulating CYP2E1/Nrf2 and TLR4/NF-B [159], scavenging mitochondrial and intracellular ROS [160], and facilitating n-butyric acid generation for homeostatic regulation of the gutliver axis [161]

It was evident that UQ oral supplementation alleviated the endothelial dysfunction and the cardiac contractility (Peres et al., 2017)

In: FalusA, editor

Bebber: Writing review & editing, Writing original draft, chapter 4.1

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