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Especially under ischemiareperfusion conditions, reducing the level of acylcarnitine has a significant effect on the energy metabolism of the heart and the maturity and proliferation of cardiomyocytes because reducing the utilization of fatty acids and increasing the reliance on glucose oxidation as an energy source can promote the recovery of myocardial function at a lower rate of oxygen consumption

NAC provides cysteine, one of the three amino acids that makes up GSH

Recent data suggest bacteria employing strategies to reprogram their virulence and to adjust the activity of virulence factors to changing conditions 6

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murinus ) or their beneficial metabolites (e.g., capsiate, CAT) can exert protective effects by inducing IL-10 secretion[30] or upregulating glutathione peroxidase 4 (GPX4) to inhibit ferroptosis[21]