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A combination of cisplatin with mTOR inhibitors causes an additive effect of cell death, and GALNT14, a member of the family of acetyl-galactosyltransferases, regulates the stability of EGFR proteins to inhibit the EGFR/mTOR pathway, thereby downregulating self-induced ferroptosis in ovarian cancer cells (Li H.W

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and (iii) vitamin B12 is essential for normal homocysteine metabolism, thereby regenerating methionine and facilitating its cycle upstream of GSH synthesis (see Figure 1)