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What to do instead: Support glutathione production directly and address sources of oxidative stress first
Mitochondria-targeted antioxidants, including MitoQ and SS31, may serve as potential therapeutic agents that reduce free radicals, maintain mitochondrial quality and function, enhance autophagy and mitophagy, and help cells remove damaged mitochondria [24, 31]
Metab Brain Dis 28(2):167174
doi: 10.1124/mol.108.048611 52 IsaiasI
Hyperglycemia-associated chromatin changes have also been connected to increased MMP-9 expression, plausibly predisposing to endothelial injury [1237]
