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Sikirics first published findings appeared in the Journal of Physiology, Paris in 1993
Simulations for alternative monothiol mechanisms (i) and (ii) from Fig
Mechanistically, NOX2/ROS induces endothelial-derived ET-1 overexpression, which activates the ERK1/2 signaling pathway to mediate the shift of microglia activation to a pro-inflammatory M1 phenotype that triggers the release of inflammatory mediators
Andrews McMeel Publishing
