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TZ slows or prevents neuronal loss, increases dopamine levels and partially restores motor function in MPTP mouse model, 6-OHDA rat model, and rotenone Drosophila model of PD, by potentiating PGK1 activity, enhancing glycolysis, and increasing cellular ATP levels

Therefore, more in vivo and in vitro experiments are still needed (Figure 1)

This mechanism makes it difficult for bacteria to develop resistance to LL-37

J Trauma Dissociation
