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However, whether distinct astrocyte subtypes regulate NPP through differential synaptic plasticity changes mediated by modulating purinergic signaling pathways remains to be further clarified

[DOI] [PubMed] [Google Scholar] 1276.Thorns V, Hansen L, Masliah E

These findings indicate that myelin repair represents a therapeutic opportunity for AD, informing a new direction and therapy basis for therapeutic interventions

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The beta cell-specific genetic risk score, composed of 30 beta cell function SNPs, was strongly associated with increasing beta cell dysfunction from prediabetes to diabetes, reinforcing the idea that beta cell failure is a primary driver of disease progression