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In particular, the astrocytic ApoE (complexed with cholesterol) promotes synaptogenesis (Mauch et al., 2001), with ApoE accumulating near the synapses (Konings et al., 2021)

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Notably, aligned with reduced steatohepatitis, our histopathological and immunofluorescence analyses coupled with biochemical verification revealed that lowering oxalate overproduction in MASH via AGXT overexpression or pharmacological inhibition of LDHA and GO potently reduces hepatic stellate cells and fibrosis, a main determinant of liver-related events and mortality in MASH 50

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