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Taken together, hyperhomocysteinemia triggers the development and progression of PD by different mechanisms, including oxidative stress, mitochondrial dysfunction, apoptosis, and endothelial dysfunction [Fig

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10.3390/biom12111702 109 MaX.WangJ.QuanQ.ZhangH.TianY.WangL.et al (2024)

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doi: 10.1016/j.chembiol.2008.02.010 145 YangW

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