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Importantly, both NOX1- and NOX2-produced ROS are required for the activation and differentiation of monocytes into proinflammatory M1 and anti-inflammatory M2 macrophages 77

In analogy, genetic ablation of HO-1 amplified the development of atherosclerotic lesions and promoted their progression, thus confirming the protective role of HO-1 against atherogenesis (181)

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PMID: 35067718 Stem CellsDUSP5 promotes osteogenic differentiation through SCP1/2-dependent phosphorylation of SMAD1