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Hemizygous deletions of 22q11DS genes were accompanied by haploinsufficiency of mitochondrial large ribosomal subunit protein 40 and led to dysregulation of short-term potentiation via impaired calcium homeostasis in mitochondria [213]

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In addition to altered carbohydrate metabolism, induction of cell death, and changes in mitochondrial permeability transition, these metabolism-related changes can lead to modulation of epigenetic regulation of gene expression