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L-2-oxothiazolidine-4-carboxylic acid (OTC) treatment attenuated plasma and tissue P-selectin levels, suggesting that the upregulation of P-selectin in hyperhomocysteinemic mice is also regulated by redox mechanisms [61]
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The authors also showed that H19 was a direct target of miR-196a and was associated with COL1A1 expression by sponging miR-196a
