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The Benoist 2014 study used the HGF/MET dimerization antagonist Hinge to block Dihexas procognitive activity in scopolamine-impaired rats, with oral Dihexa (2 mg/kg/day) reversing the scopolamine-induced Morris water maze deficit when administered alone but losing effect when co-administered with Hinge [3]

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The GSSG concentration was also significantly higher in alcoholics (8.2 0.3% of total) and nonalcoholics (8.5 0.8% of total) than in control subjects (4.4 0.2% of total), reflecting an abnormal balance between GSH and GSSG in these patients