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M.ShankarB.ZouY.MoralesJ

Acting as a damage-associated molecular pattern, ox-LDL not only triggers an innate immune response but also stimulates excessive production of reactive oxygen species (ROS), creating a pathogenic feed-forward loop (48)

However, these data were challenged by the failure of CsA to prevent hypertrophy in several models of hypertension or pressure overload (16, 35, 61)

Thus, the vitamin E supplements in the Womens Health Study probably acted as an anti-thrombotic agent, resulting in a reduction of clot formation by suppression of vitamin K activity [118]