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By activating GLP-1 and GIP receptors, it enhances insulin secretion (when glucose is present) and suppresses appetite, similar to existing incretin therapies [7] [8]
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Patients diagnosed with pernicious anaemia require lifelong treatment, as this is a chronic autoimmune condition
It's the difference between maintaining collagen synthesis signaling throughout the remodeling cycle versus spiking copper-GHK plasma levels for 36 hours and then losing the signal entirely
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