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Lanosterol has been found to inhibit transmembrane protein 175 (TMEM175), a lysosomal proton export channel, and its clearance via CYP51A1-dependent metabolism restores TMEM175 function, thereby dampening lysosomal alkalinization and mitigating the induction of alkaliptosis [68]

Ghosh S, Gregory D, Smith A, Kobzik L

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Dietary LP20 alleviated the inflammatory features caused by DLM toxicity and A

SalazarKEspinozaFCerda-GallardoGFerradaLMagdalenaRRamrezEet alSVCT2 overexpression and ascorbic acid uptake increase cortical neuron differentiation, which is dependent on vitamin C recycling between neurons and astrocytes