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28 Excessive ROS production leads to an oxidative inactivation of anti-proteases, alveolar epithelium damage, increased neutrophil retention and increased expression of various inflammatory mediators in the pulmonary microcirculation, aggravating the development of COPD

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Schisandrin-treated cells showed lower ROS levels and preservation of mitochondrial membrane potential even after glutamate exposure, compared to untreated cells (58)