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however there have been prior reports of CrAT activity outside of the mitochondria, as well as observations consistent with such activity

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It is likely that neuropathic involvement could represent a major determinant of therapeutic failure, as diabetic polyneuropathy and cardiovascular autonomic neuropathy impair nitric oxide bioavailability, endothelial responsiveness, and neural transmission within the erectile pathway

NeuroToxicology (2020) 79:204

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